However, this does not necessarily represent the policy of the Department of Education, and you should not assume endorsement by the Federal Government. For more information about the Academy please visit our website at www. View as PDF. Editor's Note: This article is a term paper which the author wrote while a student at the Northwestern University Prosthetic-Orthotic Center. The quality of the paper is an attribute to the new generation of orthotists and prosthetists.
Create a free QxMD account to take advantage of the features offered by Read like saving your papers and creating collections. There are a range of therapeutic options for PLP, both pharmacological gabapentin, amitriptyline, tricyclic antidepressants, etc and non-pharmacological transcutaneous electrical nerve stimulation, hypnosis, acupuncture, etc. A widely accepted hypothesis considers PLP to be the consequence of postamputation cortical reorganisation. New treatment approaches, such as mirror therapy MT , have been developed as a result of Ramachandran's groundbreaking research in the s. It is a valid, simple, and inexpensive treatment for PLP. The methodological quality of most publications in this field is very limited, highlighting the need for additional, high-quality studies to develop clinical protocols that could maximise the benefits of MT for patients with PLP. You are not logged in.
Military Medical Research volume 5 , Article number: 1 Cite this article. Metrics details. The prevalence of limb amputation is increasing globally as a devastating experience that can physically and psychologically affect the lifestyle of a person. The residual limb pain and phantom limb pain are common disabling sequelae after amputation surgery.
This study presents a review of the literature on the attributes and potential mechanisms involved in phantom limb pain, encompassing studies describing pain in the residual limb, phantom sensation and phantom limb pain, and the difficulties that may arise when making these distinctions. A variety of theories have been proposed to explain causal mechanisms for phantom limb pain. Conceptually, research into phantom limb pain is informed by the particular theory of chronic pain that is dominant at the time the research is undertaken. For example, early physiological theories on the etiology of phantom limb pain were grounded in specificity or pattern theories of pain. Later physiological research was based on the framework provided by Gate Control Theory and focused on identifying peripheral, spinal, and central neural mechanisms.